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The peptide stimulates endogenous GH production rather than suppressing it, so the hypothalamic-pituitary axis is not shut down in the way it would be with exogenous hormone administration
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One possible explanation is that low serum vitamin B12 levels would trap folate as 5-methyltetrahydrofolate, prevent the generation of methionine from homocysteine, and therefore reduce protein synthesis and lean tissue deposition
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