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Other studies have examined the cellular mechanisms of the relationship between UV and AMD on adult human retinal pigment epithelial cells, and EGCG has been found to exert its therapeutic role in AMD by affecting oxidative stress and apoptosis pathways such as JNK1/c-Jun pathway, extracellular signal-regulated kinase (ERK), and c-jun-NH2 terminal kinase (JNK) [129,130]