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En estudios se ha observado que el BPC-157 puede: estimular angiogénesis (nuevos vasos sanguíneos) aumentar fibroblastos (células de reparación) mejorar la síntesis de colágeno modular la inflamación sin bloquearla completamente favorecer rutas asociadas al IGF-1 Estos mecanismos explican por qué acelera tanto la recuperación

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BPC-157’s demonstrated anti-apoptotic effects through Akt/Bcl-2 upregulation and caspase inhibition in CNS models provide mechanistic rationale for RGC protection studies

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