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Notably, the interaction between AtSNX1 and the membrane was relatively weaker than that of MmSNX1, indicating lower attractive forces between AtSNX1 and the membrane compared to those between MmSNX1 and the membrane (Supplementary Fig

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Potent and clinically approved inhibitors of thymidylate and purine biosynthesis include pralatrexate and 5-fluorouracil (5-FU)