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Activation of the endothelium in sepsis occurs directly by recognition of pathogen-associated molecular patterns (PAMPs) through patron recognition receptors, such as Toll-like receptors (TLRs) expressed in ECs, and, indirectly, by released proinflammatory cytokines, such as TNF-α, IL-6, and IL-1, complement components or neutrophil extracellular traps (NETs) (99)
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