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Also, the increase of GSTA1 by bicyclol treatment resulted in a decrease in FABP1 levels and an improvement of the disease, highlighting a possible mechanism of action of GSTA1 in metabolic diseases

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In this Review, we focus on advances in our understanding of inhibition of the HGF/c-MET pathway in patients with gastro-intestinal cancers, the prominent challenges facing the clinical translation and implementation of agents targeting HGF/c-MET, and discuss the various efforts, and associated obstacles to the discovery and validation of biomarkers that will enable patient stratification in this context