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It can not only inhibit the secretion of IFN-g and IL-2 by Th1 cells, but also downregulate the secretion of IL-4 and IL-5 by Th2 cells, thereby reducing the immunopathology induced by these inflammatory factors
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Recently, the existence of an M3 activation phenotype acquired by proliferative microglia in response to colony-stimulating factor-1 (CSF-1) and IL-34 has also been suggested (Walker and Lue, 2015)
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