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As a result, downstream AKT, mTORC1, and MAPK signaling is markedly suppressed, shifting cardiomyocytes toward a pro-apoptotic state characterized by myofibrillar loss and adverse structural remodeling

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Elevated Oxidative Stress and Decreased Antioxidant Function in the Human Hippocampus and Frontal Cortex with Increasing Age: Implications for Neurodegeneration in Alzheimer’s Disease