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With acetaminophen toxicity, cellular glutathione is depleted resulting of the accumulation of APAP adducts, mainly with mitochondrial protein, inducing oxidative stress and mitochondrial injuries that lead to centrilobular hepatocyte necrosis and liver failure 3,4,5,6
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Alzheimer’s disease (AD) is characterized by the presence of amyloid-beta (Aβ) plaques and neurofibrillary tangles within the intracellular environment, neuronal death, and synaptic loss
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