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This suggests that the co-treatment with ferrous iron again switched the signaling mechanisms and mode of cell death from mitochondrial dysfunction-dependent necrosis after APAP alone to LPO-dominated ferroptosis after APAP + Fe 2+ (Figure 1)
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In a specific subtype of CRC, characterized by microsatellite instability, miR-181a, miR-135a and miR-302c were all downregulated in tissue samples and cell lines with respect to microsatellite stable samples (102)
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