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In a salicylate-induced tinnitus rat model, chronic systemic salicylate administration significantly upregulated BDNF expression and CREB phosphorylation in the AC, accompanied by ultrastructural synaptic changes (Yi et al., 2018)

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These molecular events stimulate fibroblast proliferation and differentiation into myofibroblasts, a specialized subtype capable of contraction, which play a crucial role in the wound healing process [43]